Background: This study aimed at developing a new set of maurocalcine-derived cell-penetrating peptides from truncation.

Background: Human β-cell-derived (BCD) cells can be expanded in vitro in a process involving dedifferentiation and activation of the NOTCH pathway.

Background: HIV-1 Tat engages αvβ3, leading to endothelial cell (EC) proangiogenic activation.

Background: Vasculature is a key component of the brain neurogenic stem cell niche.

Background: Integrin αv promotes melanoma cell survival in in vivo and in vitro three-dimensional environments.

Background: Polyglutamine aggregates can be internalized by mammalian cells and gain access to the cytoplasmic compartment, but the properties of the aggregates and cell surface that mediate these processes are unknown.

Background: How the adaptive ER stress response causes cell death remains enigmatic.

Background: The process of matrix degradation is an integral component of cell invasion.

Background: SALL4 is a key stem cell transcription factor that transactivates OCT4.

Background: Regulation of integrin activation has important implications for tumor cell invasion and metastasis.

Background: Store-operated Ca2+ entry is important for cell migration.

Background: Strategies on the basis of doxycycline-inducible lentiviruses in mouse cells allowed the examination of mechanisms governing somatic cell reprogramming.

Background: Rad9, a cell cycle checkpoint and DNA repair protein, is functionally related to human prostate tumorigenesis.

Background: Mechanisms underlying the cell surface export of G protein-coupled receptors (GPCRs) are poorly understood.

Background: Microtubule effects on cell migration are poorly understood.

Background: Phosphoregulation of focal adhesion (FA) proteins regulates FA dynamics underlying cell migration.

Background: Neuregulin (NRG) is overexpressed in 30% of breast cancers and mediates tumor cell migration and invasion.

Background: ELMO and DOCK180 synergize to promote Rac-dependent cell migration.

Background: V-ATPase is proposed as tumor target, but information on cell death-inducing mechanisms is rare.

Background: RAP80, a component of the BRCA1-A complex, is crucial in the cell cycle checkpoint and DNA damage repair.